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J Transl Med. 2024 Jul 26;22(1):682. doi: 10.1186/s12967-024-05399-x.
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Fatty Acid Oxidation-Glycolysis Metabolic Transition Affects ECM Homeostasis in Silica-Induced Pulmonary Fibrosis.
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Mechanics-activated fibroblasts promote pulmonary group 2 innate lymphoid cell plasticity propelling silicosis progression.
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Exosomal miRNA reprogramming in pyroptotic macrophage drives silica-induced fibroblast-to-myofibroblast transition and pulmonary fibrosis.
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The interplay of Cxcl10(+)/Mmp14(+) monocytes and Ccl3(+) neutrophils proactively mediates silica-induced pulmonary fibrosis.
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